Lesson · Foundations
Reading the basic ECG: waves, segments, intervals
1

Introduction

An ECG is a recording of the heart's electrical activity, projected through the body to electrodes on the skin. Each heartbeat produces a sequence of waves separated by segments and bounded by intervals. The shape, size, timing, and direction of these features tell you what the heart is doing — and what's wrong with it.

This lesson covers the foundational building blocks: P wave, QRS complex, T wave, the PR and QT intervals, and the PR and ST segments. By the end, you will be able to identify each feature on a normal sinus rhythm strip, measure intervals correctly, and understand what each represents physiologically.

Clinical pearl
Throughout this lesson, the term "lead II" refers to the standard rhythm-strip view from the right arm to the left leg. Most teaching examples use lead II because the heart's electrical vector aligns closely with it, producing the largest and clearest deflections.
PQRSTPRST25 mm/s · 10 mm/mV · Lead II
Normal sinus rhythm in lead II — the elements you'll learn to identify in this lesson.
2

The P wave

The P wave represents atrial depolarisation — the electrical activation that spreads from the sinoatrial (SA) node across the right and left atria. The SA node sits at the top of the right atrium, and the depolarisation wave moves leftward and downward, which is why the P wave is upright in leads I and II in normal sinus rhythm.

A normal P wave is small (less than 2.5 mm tall and less than 110 ms wide), smoothly rounded, and precedes every QRS complex. Its presence tells you the rhythm originates from the atria; its shape tells you whether the SA node or another atrial focus is firing.

Clinical pearl
The P wave is best assessed in lead II, where atrial depolarisation projects most strongly. If you can't find P waves in lead II, check V1 — biphasic P waves there are common and diagnostic of left atrial enlargement.
Watch out
A "P wave" that appears in lead II but is negative in someone who should be in sinus rhythm is a major red flag — most often LA-RA electrode reversal, occasionally an ectopic atrial focus or junctional rhythm with retrograde conduction. Always recheck the placement before interpreting.
Quick check
In a healthy adult in sinus rhythm, the P wave in lead II should be:
3

The QRS complex

The QRS complex represents ventricular depolarisation — the rapid spread of electrical activity through the His-Purkinje system and into the ventricular myocardium. Because the ventricles have far more muscle mass than the atria, the QRS deflection is much larger than the P wave.

By convention, the first downward deflection after the P wave is called Q, the first upward deflection is R, and the downward deflection following R is S. Not every complex has all three components; lead V1, for example, often has a tiny R followed by a deep S (rS pattern), while V6 typically has a tall R with a small S.

A normal QRS lasts less than 100 ms (less than 2.5 small boxes). When the QRS is wider than 120 ms, the activation is no longer occurring through the normal conduction pathway — possibilities include bundle branch block, ventricular rhythm, pre-excitation (WPW), hyperkalaemia, or sodium-channel blocker effect.

Try itQRS duration
QRS25 mm/s · 10 mm/mV · Lead II
QRS duration90ms
Drag the slider to widen the QRS and watch the morphology change. Anything beyond 120 ms is pathological.
Clinical pearl
The shape of the QRS in V1 is a powerful clue. A tall R wave (R greater than S) suggests right ventricular hypertrophy, posterior MI, RBBB, or WPW. A deep S with no R wave (QS pattern) suggests prior anterior MI or LBBB. Always look at V1 first when interpreting morphology.
4

The T wave

The T wave represents ventricular repolarisation — the recovery of the ventricles after depolarisation. Repolarisation is a slower process than depolarisation, which is why the T wave is broader and lower-amplitude than the QRS.

Normal T waves are upright in leads I, II, V3 through V6, and inverted in aVR (always) and often in V1. Asymmetric in shape — the upstroke is more gradual than the downstroke — and roughly one-third the height of the R wave that precedes it.

Watch out
T-wave inversion in leads where T should be upright (I, II, V3-V6) suggests ischaemia, strain, hypokalaemia, raised intracranial pressure, pulmonary embolism, or several other pathologies. The pattern of inversion (which leads, depth, symmetry) and the clinical context determine the differential.
Quick check
In which lead is T-wave inversion always normal?
5

Intervals: PR, QRS, QT

Three intervals matter clinically. They measure the time taken for different parts of the cardiac electrical cycle and are central to identifying conduction abnormalities, drug effects, and electrolyte disturbances.

IntervalFromToNormalIf abnormal
PR intervalStart of PStart of QRS120–200 msProlonged: 1° AV block · Short with delta wave: WPW
QRS durationStart of QRSEnd of QRS<100 ms≥120 ms: BBB, ventricular rhythm, hyperkalaemia, drug toxicity
QT intervalStart of QRSEnd of TQTc <450 ms (m), <460 ms (f)>500 ms substantially raises torsades risk

PR interval — measured from the start of the P wave to the start of the QRS — represents conduction from atria through the AV node and His-Purkinje system to the ventricles. Prolonged PR (greater than 200 ms) is first-degree AV block; very short PR with a slurred QRS upstroke is pre-excitation (WPW).

QRS duration — from start to end of QRS — measures ventricular depolarisation time. Normal less than 100 ms.

QT interval — start of QRS to end of T — measures total ventricular depolarisation plus repolarisation. The QT shortens at faster heart rates, so it's corrected using Bazett's formula (QTc = QT divided by the square root of RR in seconds). QTc longer than 460 ms (women) or 450 ms (men) is prolonged; longer than 500 ms substantially raises torsades risk.

Clinical pearl
Each small box on standard ECG paper is 40 ms; each large box is 200 ms. A PR interval spanning 5 small boxes is exactly 200 ms — at the upper limit of normal. Train yourself to "see" boxes rather than reach for callipers for routine measurements.
Try itPR interval
PR25 mm/s · 10 mm/mV · Lead II
PR interval160ms
Upper limit of normal: 200ms
Drag to lengthen the PR interval. Beyond 200 ms (5 small boxes) is first-degree AV block.
Try this
Practice measuring intervals
The workbench has a calliper tool — open any rhythm strip, click two points, and the interval is calculated for you in milliseconds and small boxes.
Open the workbench
6

Segments: PR and ST

Segments are the flat baseline portions between waves. They serve as the visual reference for detecting elevation or depression elsewhere in the trace.

The PR segment — between the end of the P wave and the start of QRS — is the electrical baseline reference. Use it (not the TP segment, not the start of the strip) to judge whether ST is elevated or depressed.

The ST segment — between the end of QRS (the J point) and the start of T — is the most clinically charged piece of the ECG. ST elevation in a coronary territory is a STEMI until proven otherwise; ST depression suggests subendocardial ischaemia, posterior MI (mirror-image), or non-coronary causes.

Clinical pearl
ST elevation is measured at the J point (the junction of QRS and ST) relative to the PR segment. Significant elevation is at least 1 mm in two contiguous limb leads, or at least 2 mm in V2-V3 (men over 40), or at least 1.5 mm in V2-V3 (women).
Try itST shift
ST25 mm/s · 10 mm/mV · Lead II
ST shift0.00mV
Significant elevation: 0.1mV
Drag positive for ST elevation, negative for depression. Watch how the segment between QRS and T moves relative to the baseline.
Watch out
Not all ST elevation is STEMI. Pericarditis produces diffuse concave-upward elevation with PR depression. Early repolarisation, common in young athletes, mimics STEMI but is stable over time. Left bundle branch block obscures STEMI criteria entirely — Sgarbossa's criteria help.
7

Summary & next steps

You now have the vocabulary to describe a normal ECG: P, Q, R, S, T waves, PR and ST segments, and PR, QRS, QT intervals. You know what each represents physiologically and what abnormal values suggest.

The next lesson — Heart rate and rhythm interpretation — applies these concepts to recognising sinus rhythm, sinus tachycardia and bradycardia, and the irregularly irregular rhythm of atrial fibrillation. After that, axis determination, then chamber enlargement, then ischaemia patterns.

Lesson complete
Mark this lesson as complete to move to the next foundational topic. Your spaced-repetition schedule will surface relevant questions over the coming days to lock in what you've learned.
Educational use only — not for clinical interpretation or patient care.